USP39 promotes hepatocellular carcinogenesis through regulating alternative splicing in cooperation with SRSF6/HNRNPC

Abnormal alternative splicing (AS) caused by alterations in spliceosomal factors is implicated in cancers. Standard models posit that splice site selection is mainly determined by early spliceosomal U1 and U2 snRNPs. Whether and how other mid/late-acting spliceosome components such as USP39 modulate...

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Detalles Bibliográficos
Autores principales: Zheng, Jingyi, Wu, Shasha, Tang, Mao, Xi, Shaoyan, Wang, Yanchen, Ren, Jun, Luo, Hao, Hu, Pengchao, Sun, Liangzhan, Du, Yuyang, Yang, Hui, Wang, Fenfen, Gao, Han, Dai, Ziwei, Ou, Xijun, Li, Yan
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Nature Publishing Group UK 2023
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10567755/
https://www.ncbi.nlm.nih.gov/pubmed/37821439
http://dx.doi.org/10.1038/s41419-023-06210-3

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10567755/
https://www.ncbi.nlm.nih.gov/pubmed/37821439
http://dx.doi.org/10.1038/s41419-023-06210-3

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