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Blood flow, not hypoxia, determines intramucosal PCO(2)
Monitoring tissue hypoxia in critically ill patients is a challenging task. Tissue PCO(2 )has long been proposed as a marker of tissue hypoxia, although there is considerable controversy on whether the rise in CO(2 )with hypoxia is caused by anaerobic metabolism and excess CO(2 )production or by the...
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| Formato: | Texto |
| Lenguaje: | English |
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BioMed Central
2005
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| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1175940/ https://www.ncbi.nlm.nih.gov/pubmed/15774068 http://dx.doi.org/10.1186/cc3489 |