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Neuropathic pain develops normally in mice lacking both Na(v)1.7 and Na(v)1.8

Two voltage gated sodium channel α-subunits, Na(v)1.7 and Na(v)1.8, are expressed at high levels in nociceptor terminals and have been implicated in the development of inflammatory pain. Mis-expression of voltage-gated sodium channels by damaged sensory neurons has also been implicated in the develo...

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Detalles Bibliográficos
Autores principales: Nassar, Mohammed A, Levato, Alessandra, Stirling, L Caroline, Wood, John N
Formato: Texto
Lenguaje:English
Publicado: BioMed Central 2005
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1215513/
https://www.ncbi.nlm.nih.gov/pubmed/16111501
http://dx.doi.org/10.1186/1744-8069-1-24