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Neuropathic pain develops normally in mice lacking both Na(v)1.7 and Na(v)1.8
Two voltage gated sodium channel α-subunits, Na(v)1.7 and Na(v)1.8, are expressed at high levels in nociceptor terminals and have been implicated in the development of inflammatory pain. Mis-expression of voltage-gated sodium channels by damaged sensory neurons has also been implicated in the develo...
| Autores principales: | , , , |
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| Formato: | Texto |
| Lenguaje: | English |
| Publicado: |
BioMed Central
2005
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| Materias: | |
| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1215513/ https://www.ncbi.nlm.nih.gov/pubmed/16111501 http://dx.doi.org/10.1186/1744-8069-1-24 |