Inverted repeat-stimulated sister-chromatid exchange events are RAD1-independent but reduced in a msh2 mutant

Inverted repeats (IRs) and trinucleotide repeats (TNRs) that have the potential to form secondary structures in vivo are known to cause genome rearrangements. Expansions of TNRs in humans are associated with several neurological disorders. Both IRs and TNRs stimulate spontaneous unequal sister-chrom...

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Detalles Bibliográficos
Autores principales: Nag, Dilip K., Fasullo, Michael, Dong, Zheng, Tronnes, Ashlie
Formato: Texto
Lenguaje:English
Publicado: Oxford University Press 2005
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1216339/
https://www.ncbi.nlm.nih.gov/pubmed/16166656
http://dx.doi.org/10.1093/nar/gki835

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1216339/
https://www.ncbi.nlm.nih.gov/pubmed/16166656
http://dx.doi.org/10.1093/nar/gki835

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