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Inverted repeat-stimulated sister-chromatid exchange events are RAD1-independent but reduced in a msh2 mutant
Inverted repeats (IRs) and trinucleotide repeats (TNRs) that have the potential to form secondary structures in vivo are known to cause genome rearrangements. Expansions of TNRs in humans are associated with several neurological disorders. Both IRs and TNRs stimulate spontaneous unequal sister-chrom...
Autores principales: | Nag, Dilip K., Fasullo, Michael, Dong, Zheng, Tronnes, Ashlie |
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Formato: | Texto |
Lenguaje: | English |
Publicado: |
Oxford University Press
2005
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1216339/ https://www.ncbi.nlm.nih.gov/pubmed/16166656 http://dx.doi.org/10.1093/nar/gki835 |
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