Inhibition of p38 mitogen-activated protein kinase enhances c-Jun N-terminal kinase activity: Implication in inducible nitric oxide synthase expression

BACKGROUND: Nitric oxide (NO) is an inflammatory mediator, which acts as a cytotoxic agent and modulates immune responses and inflammation. p38 mitogen-activated protein kinase (MAPK) signal transduction pathway is activated by chemical and physical stress and regulates immune responses. Previous st...

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Detalles Bibliográficos
Autores principales: Lahti, Aleksi, Sareila, Outi, Kankaanranta, Hannu, Moilanen, Eeva
Formato: Texto
Lenguaje:English
Publicado: BioMed Central 2006
Materias:
Research Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1402273/
https://www.ncbi.nlm.nih.gov/pubmed/16504051
http://dx.doi.org/10.1186/1471-2210-6-5

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1402273/
https://www.ncbi.nlm.nih.gov/pubmed/16504051
http://dx.doi.org/10.1186/1471-2210-6-5

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