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Inhibition of p38 mitogen-activated protein kinase enhances c-Jun N-terminal kinase activity: Implication in inducible nitric oxide synthase expression

BACKGROUND: Nitric oxide (NO) is an inflammatory mediator, which acts as a cytotoxic agent and modulates immune responses and inflammation. p38 mitogen-activated protein kinase (MAPK) signal transduction pathway is activated by chemical and physical stress and regulates immune responses. Previous st...

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Detalles Bibliográficos
Autores principales:	Lahti, Aleksi, Sareila, Outi, Kankaanranta, Hannu, Moilanen, Eeva
Formato:	Texto
Lenguaje:	English
Publicado:	BioMed Central 2006
Materias:	Research Article
Acceso en línea:	https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1402273/ https://www.ncbi.nlm.nih.gov/pubmed/16504051 http://dx.doi.org/10.1186/1471-2210-6-5

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