NSAIDs Modulate CDKN2A, TP53, and DNA Content Risk for Progression to Esophageal Adenocarcinoma

BACKGROUND: Somatic genetic CDKN2A, TP53, and DNA content abnormalities are common in many human cancers and their precursors, including esophageal adenocarcinoma (EA) and Barrett's esophagus (BE), conditions for which aspirin and other nonsteroidal anti-inflammatory drugs (NSAIDs) have been pr...

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Detalles Bibliográficos
Autores principales: Galipeau, Patricia C, Li, Xiaohong, Blount, Patricia L, Maley, Carlo C, Sanchez, Carissa A, Odze, Robert D, Ayub, Kamran, Rabinovitch, Peter S, Vaughan, Thomas L, Reid, Brian J
Formato: Texto
Lenguaje:English
Publicado: Public Library of Science 2007
Materias:
Research Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1808095/
https://www.ncbi.nlm.nih.gov/pubmed/17326708
http://dx.doi.org/10.1371/journal.pmed.0040067

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1808095/
https://www.ncbi.nlm.nih.gov/pubmed/17326708
http://dx.doi.org/10.1371/journal.pmed.0040067

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