Cockayne syndrome B protein stimulates apurinic endonuclease 1 activity and protects against agents that introduce base excision repair intermediates

The Cockayne syndrome B (CSB) protein—defective in a majority of patients suffering from the rare autosomal disorder CS—is a member of the SWI2/SNF2 family with roles in DNA repair and transcription. We demonstrate herein that purified recombinant CSB and the major human apurinic/apyrimidinic (AP) e...

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Detalles Bibliográficos
Autores principales: Wong, Heng-Kuan, Muftuoglu, Meltem, Beck, Gad, Imam, Syed Z., Bohr, Vilhelm A., Wilson, David M.
Formato: Texto
Lenguaje:English
Publicado: Oxford University Press 2007
Materias:
Nucleic Acid Enzymes
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1919475/
https://www.ncbi.nlm.nih.gov/pubmed/17567611
http://dx.doi.org/10.1093/nar/gkm404

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1919475/
https://www.ncbi.nlm.nih.gov/pubmed/17567611
http://dx.doi.org/10.1093/nar/gkm404

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