Modulation of microglial/macrophage activation by macrophage inhibitory factor (TKP) or tuftsin (TKPR) attenuates the disease course of experimental autoimmune encephalomyelitis

BACKGROUND: Myelin Oligodendrocyte Glycoprotein (MOG)-induced experimental autoimmune encephalomyelitis (EAE) is the most commonly used mouse model for multiple sclerosis (MS). During the of progression of EAE, microglia, the immunocompetent cells of the brain, become activated and accumulate around...

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Detalles Bibliográficos
Autores principales: Bhasin, Madhuri, Wu, Muzhou, Tsirka, Stella E
Formato: Texto
Lenguaje:English
Publicado: BioMed Central 2007
Materias:
Research Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1937009/
https://www.ncbi.nlm.nih.gov/pubmed/17634104
http://dx.doi.org/10.1186/1471-2172-8-10

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1937009/
https://www.ncbi.nlm.nih.gov/pubmed/17634104
http://dx.doi.org/10.1186/1471-2172-8-10

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