Cargando…
Clonal variation in the sensitivity of B16 melanoma to m-AMSA.
A hypothesis that m-AMSA may have greater cytotoxicity in melanin-containing tumour tissues, because it may reversibly bind to melanin, leading to prolonged drug exposure, was examined. Clonal lines of B16 melanoma which differed widely in pigmentation level were selected by isolating artificial lun...
Autores principales: | Stephens, T. C., Peacock, J. H. |
---|---|
Formato: | Texto |
Lenguaje: | English |
Publicado: |
Nature Publishing Group
1982
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2011041/ https://www.ncbi.nlm.nih.gov/pubmed/6896455 |
Ejemplares similares
-
Enhanced killing of mammalian cells by radiation combined with m-AMSA.
por: Roberts, P. B., et al.
Publicado: (1980) -
Expression of Drug Resistance Genes in VP‐16 and mAMSA‐selected Human Carcinoma Cells
por: Matsumoto, Yoshihito, et al.
Publicado: (2001) -
A comparison of adriamycin and mAMSA in vitro: cell lethality and SCE studies.
por: West, C., et al.
Publicado: (1981) -
Resistance to 4-(9-acridinylamino) methanesulphon-m-anisidide (m-AMSA) in human myeloid leukaemia.
por: Skinner, W. L., et al.
Publicado: (1990) -
Cell yield and cell survival following chemotherapy of the B16 melanoma.
por: Stephens, T. C., et al.
Publicado: (1978)