Nitrite augments tolerance to ischemia/reperfusion injury via the modulation of mitochondrial electron transfer

Nitrite (NO(2) (−)) is an intrinsic signaling molecule that is reduced to NO during ischemia and limits apoptosis and cytotoxicity at reperfusion in the mammalian heart, liver, and brain. Although the mechanism of nitrite-mediated cytoprotection is unknown, NO is a mediator of the ischemic precondit...

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Detalles Bibliográficos
Autores principales: Shiva, Sruti, Sack, Michael N., Greer, James J., Duranski, Mark, Ringwood, Lorna A., Burwell, Lindsay, Wang, Xunde, MacArthur, Peter H., Shoja, Amir, Raghavachari, Nalini, Calvert, John W., Brookes, Paul S., Lefer, David J., Gladwin, Mark T.
Formato: Texto
Lenguaje:English
Publicado: The Rockefeller University Press 2007
Materias:
Articles
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2118713/
https://www.ncbi.nlm.nih.gov/pubmed/17682069
http://dx.doi.org/10.1084/jem.20070198

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2118713/
https://www.ncbi.nlm.nih.gov/pubmed/17682069
http://dx.doi.org/10.1084/jem.20070198

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