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Activity-induced internalization and rapid degradation of sodium channels in cultured fetal neurons
A regulatory mechanism for neuronal excitability consists in controlling sodium channel density at the plasma membrane. In cultured fetal neurons, activation of sodium channels by neurotoxins, e.g., veratridine and alpha-scorpion toxin (alpha-ScTx) that enhance the channel open state probability ind...
Formato: | Texto |
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Lenguaje: | English |
Publicado: |
The Rockefeller University Press
1996
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Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2120887/ https://www.ncbi.nlm.nih.gov/pubmed/8707833 |