Maximal HIV-1 Replication in Alveolar Macrophages during Tuberculosis Requires both Lymphocyte Contact and Cytokines

HIV-1 replication is markedly upregulated in alveolar macrophages (AM) during pulmonary tuberculosis (TB). This is associated with loss of an inhibitory CCAAT enhancer binding protein β (C/EBPβ) transcription factor and activation of nuclear factor (NF)-κB. Since the cellular immune response in pulm...

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Detalles Bibliográficos
Autores principales: Hoshino, Yoshihiko, Nakata, Koh, Hoshino, Satomi, Honda, Yoshihiro, Tse, Doris B., Shioda, Tatsuo, Rom, William N., Weiden, Michael
Formato: Texto
Lenguaje:English
Publicado: The Rockefeller University Press 2002
Materias:
Original Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2193627/
https://www.ncbi.nlm.nih.gov/pubmed/11854362
http://dx.doi.org/10.1084/jem.20011614

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2193627/
https://www.ncbi.nlm.nih.gov/pubmed/11854362
http://dx.doi.org/10.1084/jem.20011614

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