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Maximal HIV-1 Replication in Alveolar Macrophages during Tuberculosis Requires both Lymphocyte Contact and Cytokines

HIV-1 replication is markedly upregulated in alveolar macrophages (AM) during pulmonary tuberculosis (TB). This is associated with loss of an inhibitory CCAAT enhancer binding protein β (C/EBPβ) transcription factor and activation of nuclear factor (NF)-κB. Since the cellular immune response in pulm...

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Detalles Bibliográficos
Autores principales:	Hoshino, Yoshihiko, Nakata, Koh, Hoshino, Satomi, Honda, Yoshihiro, Tse, Doris B., Shioda, Tatsuo, Rom, William N., Weiden, Michael
Formato:	Texto
Lenguaje:	English
Publicado:	The Rockefeller University Press 2002
Materias:	Original Article
Acceso en línea:	https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2193627/ https://www.ncbi.nlm.nih.gov/pubmed/11854362 http://dx.doi.org/10.1084/jem.20011614

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