Immunoglobulin G–mediated Inflammatory Responses Develop Normally in Complement-deficient Mice

The role of complement in immunoglobulin G–triggered inflammation was studied in mice genetically deficient in complement components C3 and C4. Using the reverse passive Arthus reaction and experimental models of immune hemolytic anemia and immune thrombocytopenia, we show that these mice have types...

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Detalles Bibliográficos
Autores principales: Sylvestre, Diana, Clynes, Raphael, Ma, Minga, Warren, Henry, Carroll, Michael C., Ravetch, Jeffrey V.
Formato: Texto
Lenguaje:English
Publicado: The Rockefeller University Press 1996
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2196366/
https://www.ncbi.nlm.nih.gov/pubmed/8976192

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2196366/
https://www.ncbi.nlm.nih.gov/pubmed/8976192

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