Cargando…
Transgene Expression of bcl-x(L) Permits Anti-immunoglobulin (Ig)–induced Proliferation in xid B Cells
Mutations in the tyrosine kinase, Btk, result in a mild immunodeficiency in mice (xid). While B lymphocytes from xid mice do not proliferate to anti-immunoglobulin (Ig), we show here induction of the complete complement of cell cycle regulatory molecules, though the level of induction is about half...
Autores principales: | Solvason, Nanette, Wu, Wei Wei, Kabra, Nisha, Lund-Johansen, Fridjtof, Roncarolo, Maria Grazia, Behrens, Timothy W., Grillot, Didier A.M., Nunez, Gabriel, Lees, Emma, Howard, Maureen |
---|---|
Formato: | Texto |
Lenguaje: | English |
Publicado: |
The Rockefeller University Press
1998
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2212200/ https://www.ncbi.nlm.nih.gov/pubmed/9529324 |
Ejemplares similares
-
Physiology of B cells in mice with X-linked immunodeficiency (xid). III. Disappearance of xid B cells in double bone marrow chimeras
Publicado: (1984) -
VH gene family expression in mice with the xid defect
Publicado: (1991) -
Human rheumatoid factor cross-idiotypes. IV. Studies on WA XId-positive IgM without rheumatoid factor activity provide evidence that the WA XId is not unique to rheumatoid factors and is distinct from the 17.109 and G6 XIds
Publicado: (1993) -
The xid gene controls Ia.W39-associated immune response gene function
Publicado: (1981) -
X-linked immunodeficient (XID) mice exhibit high susceptibility to Cryptococcus gattii infection
por: Diniz-Lima, Israel, et al.
Publicado: (2021)