Self-assembly of human latexin into amyloid-like oligomers

BACKGROUND: In conformational disorders, it is not evident which amyloid aggregates affect specific molecular mechanisms or cellular pathways, which cause disease because of their quantity and mechanical features and which states in aggregate formation are pathogenic. Due to the increasing consensus...

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Detalles Bibliográficos
Autores principales: Pallarés, Irantzu, Berenguer, Clara, Avilés, Francesc X, Vendrell, Josep, Ventura, Salvador
Formato: Texto
Lenguaje:English
Publicado: BioMed Central 2007
Materias:
Research Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2212644/
https://www.ncbi.nlm.nih.gov/pubmed/17996039
http://dx.doi.org/10.1186/1472-6807-7-75

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2212644/
https://www.ncbi.nlm.nih.gov/pubmed/17996039
http://dx.doi.org/10.1186/1472-6807-7-75

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