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Self-assembly of human latexin into amyloid-like oligomers
BACKGROUND: In conformational disorders, it is not evident which amyloid aggregates affect specific molecular mechanisms or cellular pathways, which cause disease because of their quantity and mechanical features and which states in aggregate formation are pathogenic. Due to the increasing consensus...
| Autores principales: | , , , , |
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| Formato: | Texto |
| Lenguaje: | English |
| Publicado: |
BioMed Central
2007
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| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2212644/ https://www.ncbi.nlm.nih.gov/pubmed/17996039 http://dx.doi.org/10.1186/1472-6807-7-75 |