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Forced expression of the cell cycle inhibitor p57(Kip2 )in cardiomyocytes attenuates ischemia-reperfusion injury in the mouse heart

BACKGROUND: Myocardial hypoxic-ischemic injury is the cause of significant morbidity and mortality worldwide. The cardiomyocyte response to hypoxic-ischemic injury is known to include changes in cell cycle regulators. The cyclin-dependent kinase inhibitor p57(Kip2 )is involved in cell cycle control,...

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Detalles Bibliográficos
Autores principales:	Haley, Sheila A, Zhao, Ting, Zou, Lijun, Klysik, Jan E, Padbury, James F, Kochilas, Lazaros K
Formato:	Texto
Lenguaje:	English
Publicado:	BioMed Central 2008
Materias:	Research Article
Acceso en línea:	https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2268709/ https://www.ncbi.nlm.nih.gov/pubmed/18312674 http://dx.doi.org/10.1186/1472-6793-8-4

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