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Inhibition of endosomal sequestration of basic anticancer drugs: influence on cytotoxicity and tissue penetration

The basic drugs doxorubicin and mitoxantrone are known to be concentrated in acidic endosomes of cells. Here, we address the hypotheses that raising endosomal pH with the modifying agents chloroquine, omeprazole or bafilomycin A might decrease sequestration of anticancer drugs in endosomes, thereby...

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Detalles Bibliográficos
Autores principales: Lee, C M, Tannock, I F
Formato: Texto
Lenguaje:English
Publicado: Nature Publishing Group 2006
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2361369/
https://www.ncbi.nlm.nih.gov/pubmed/16495919
http://dx.doi.org/10.1038/sj.bjc.6603010
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author Lee, C M
Tannock, I F
author_facet Lee, C M
Tannock, I F
author_sort Lee, C M
collection PubMed
description The basic drugs doxorubicin and mitoxantrone are known to be concentrated in acidic endosomes of cells. Here, we address the hypotheses that raising endosomal pH with the modifying agents chloroquine, omeprazole or bafilomycin A might decrease sequestration of anticancer drugs in endosomes, thereby increasing their cytotoxicity and availability for tissue penetration. Chloroquine, omeprazole and bafilomycin A showed concentration-dependent effects to raise endosomal pH, and to inhibit sequestration of doxorubicin in endosomes. Chloroquine and omeprazole but not bafilomycin A decreased the net uptake of doxorubicin into cells, but there was no significant effect on uptake of mitoxantrone. Omeprazole and bafilomycin A increased the cytotoxicity of the anticancer drugs for cultured cells, as measured in a clonogenic assay, whereas chloroquine had minimal effects on cytotoxicity despite reduced uptake of doxorubicin. Omeprazole but not chloroquine or bafilomycin A increased the penetration of anticancer drugs through multicellular layers of tumour tissue. We conclude that modifiers of endosomal pH might increase therapeutic effectiveness of basic drugs by increasing their toxicity and/or tissue penetration in solid tumours.
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spelling pubmed-23613692009-09-10 Inhibition of endosomal sequestration of basic anticancer drugs: influence on cytotoxicity and tissue penetration Lee, C M Tannock, I F Br J Cancer Translational Therapeutics The basic drugs doxorubicin and mitoxantrone are known to be concentrated in acidic endosomes of cells. Here, we address the hypotheses that raising endosomal pH with the modifying agents chloroquine, omeprazole or bafilomycin A might decrease sequestration of anticancer drugs in endosomes, thereby increasing their cytotoxicity and availability for tissue penetration. Chloroquine, omeprazole and bafilomycin A showed concentration-dependent effects to raise endosomal pH, and to inhibit sequestration of doxorubicin in endosomes. Chloroquine and omeprazole but not bafilomycin A decreased the net uptake of doxorubicin into cells, but there was no significant effect on uptake of mitoxantrone. Omeprazole and bafilomycin A increased the cytotoxicity of the anticancer drugs for cultured cells, as measured in a clonogenic assay, whereas chloroquine had minimal effects on cytotoxicity despite reduced uptake of doxorubicin. Omeprazole but not chloroquine or bafilomycin A increased the penetration of anticancer drugs through multicellular layers of tumour tissue. We conclude that modifiers of endosomal pH might increase therapeutic effectiveness of basic drugs by increasing their toxicity and/or tissue penetration in solid tumours. Nature Publishing Group 2006-03-27 2006-02-21 /pmc/articles/PMC2361369/ /pubmed/16495919 http://dx.doi.org/10.1038/sj.bjc.6603010 Text en Copyright © 2006 Cancer Research UK https://creativecommons.org/licenses/by/4.0/This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made.The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material.If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit https://creativecommons.org/licenses/by/4.0/.
spellingShingle Translational Therapeutics
Lee, C M
Tannock, I F
Inhibition of endosomal sequestration of basic anticancer drugs: influence on cytotoxicity and tissue penetration
title Inhibition of endosomal sequestration of basic anticancer drugs: influence on cytotoxicity and tissue penetration
title_full Inhibition of endosomal sequestration of basic anticancer drugs: influence on cytotoxicity and tissue penetration
title_fullStr Inhibition of endosomal sequestration of basic anticancer drugs: influence on cytotoxicity and tissue penetration
title_full_unstemmed Inhibition of endosomal sequestration of basic anticancer drugs: influence on cytotoxicity and tissue penetration
title_short Inhibition of endosomal sequestration of basic anticancer drugs: influence on cytotoxicity and tissue penetration
title_sort inhibition of endosomal sequestration of basic anticancer drugs: influence on cytotoxicity and tissue penetration
topic Translational Therapeutics
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2361369/
https://www.ncbi.nlm.nih.gov/pubmed/16495919
http://dx.doi.org/10.1038/sj.bjc.6603010
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