Prion protein attenuates excitotoxicity by inhibiting NMDA receptors

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It is well established that misfolded forms of cellular prion protein (PrP [PrP(C)]) are crucial in the genesis and progression of transmissible spongiform encephalitis, whereas the function of native PrP(C) remains incompletely understood. To determine the physiological role of PrP(C), we examine t...

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Detalles Bibliográficos
Autores principales: Khosravani, Houman, Zhang, Yunfeng, Tsutsui, Shigeki, Hameed, Shahid, Altier, Christophe, Hamid, Jawed, Chen, Lina, Villemaire, Michelle, Ali, Zenobia, Jirik, Frank R., Zamponi, Gerald W.
Formato: Texto
Lenguaje:English
Publicado: The Rockefeller University Press 2008
Materias:
Research Articles
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2364707/
https://www.ncbi.nlm.nih.gov/pubmed/18443219
http://dx.doi.org/10.1083/jcb.200711002

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2364707/
https://www.ncbi.nlm.nih.gov/pubmed/18443219
http://dx.doi.org/10.1083/jcb.200711002

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