Cargando…
Fibrinogen decreases cardiomyocyte contractility through an ICAM-1-dependent mechanism
INTRODUCTION: Cardiomyocytes exposed to inflammatory processes express intracellular adhesion molecule-1 (ICAM-1). We investigated whether fibrinogen and fibrinogen degradation products, including D-dimer, could alter cardiomyocyte contractile function through interaction with ICAM-1 found on inflam...
Autores principales: | Boyd, John H, Chau, Edmond H, Tokunanga, Chiho, Bateman, Ryon M, Haljan, Greg, Davani, Ehsan Y, Wang, Yinjin, Walley, Keith R |
---|---|
Formato: | Texto |
Lenguaje: | English |
Publicado: |
BioMed Central
2008
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2374637/ https://www.ncbi.nlm.nih.gov/pubmed/18173852 http://dx.doi.org/10.1186/cc6213 |
Ejemplares similares
-
Microvascular resuscitation as a therapeutic goal in severe sepsis
por: Bateman, Ryon M, et al.
Publicado: (2005) -
Iron deficiency impairs contractility of human cardiomyocytes through decreased mitochondrial function
por: Hoes, Martijn F., et al.
Publicado: (2018) -
Fractalkine Depresses Cardiomyocyte Contractility
por: Taube, David, et al.
Publicado: (2013) -
S-Nitrosoglutathione Acts as a Small Molecule Modulator of Human Fibrin Clot Architecture
por: Bateman, Ryon M., et al.
Publicado: (2012) -
Correction: Fractalkine Depresses Cardiomyocyte Contractility
por: Taube, David, et al.
Publicado: (2013)