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IGF-1 regulates cAMP levels in astrocytes through a β(2)-adrenergic receptor-dependant mechanism

We have recently demonstrated that neonatal astrocytes derived from mice lacking beta-2 adrenergic receptors (β(2)AR) possess higher proliferation rates, as compared to wild-type cells, an attribute that was shown to involve insulin-like growth factor (IGF) signaling. In the present study, we demons...

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Detalles Bibliográficos
Autores principales: Chesik, Daniel, Wilczak, Nadine, De Keyser, Jacques
Formato: Texto
Lenguaje:English
Publicado: Ivyspring International Publisher 2008
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2500150/
https://www.ncbi.nlm.nih.gov/pubmed/18690292