Cargando…
Essential contribution of CCL3 to alkali-induced corneal neovascularization by regulating vascular endothelial growth factor production by macrophages
PURPOSE: To evaluate the roles of CCL3 and its specific chemokine receptors, CCR1 and CCR5, in alkali-induced corneal neovascularization (CNV). METHODS: Chemical denudation of corneal and limbal epithelium was performed on wild-type (WT) BALB/c mice and CCL3-, CCR1-, and CCR5-deficienct (knockout [K...
Autores principales: | Lu, Peirong, Li, Longbiao, Wu, Yu, Mukaida, Naofumi, Zhang, Xueguang |
---|---|
Formato: | Texto |
Lenguaje: | English |
Publicado: |
Molecular Vision
2008
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2529469/ https://www.ncbi.nlm.nih.gov/pubmed/18776949 |
Ejemplares similares
-
Critical role of SDF-1α-induced progenitor cell recruitment and macrophage VEGF production in the experimental corneal neovascularization
por: Liu, Gaoqin, et al.
Publicado: (2011) -
ADP-ribosylation factor as a novel target for corneal neovascularization regression
por: Dai, Chunyan, et al.
Publicado: (2012) -
Inhibition of PDGF-BB reduces alkali-induced corneal neovascularization in mice
por: Chen, Lei, et al.
Publicado: (2021) -
Role of macrophage inflammatory protein (MIP)-1α/CCL3 in leukemogenesis
por: Baba, Tomohisa, et al.
Publicado: (2014) -
Streptozotocin-induced diabetic mice exhibit reduced experimental choroidal neovascularization but not corneal neovascularization
por: Liu, Gaoqin, et al.
Publicado: (2018)