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Inhibition of HIV-1 gene expression by Sam68ΔC: multiple targets but a common mechanism?
Two recent publications have explored the mechanisms by which a mutant of the host protein Sam68 blocks HIV-1 structural protein synthesis and expands its activity to encompass Nef. Although the two studies propose different mechanisms for the responses observed, it is possible that a common activit...
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| Formato: | Texto |
| Lenguaje: | English |
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BioMed Central
2009
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| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2651844/ https://www.ncbi.nlm.nih.gov/pubmed/19254361 http://dx.doi.org/10.1186/1742-4690-6-22 |