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β-Arrestin-2 Mediates Anti-apoptotic Signaling through Regulation of BAD Phosphorylation
β-Arrestins, originally discovered as terminators of G protein-coupled receptor signaling, have more recently been appreciated to also function as signal transducers in their own right, although the consequences for cellular physiology have not been well understood. Here we demonstrate that β-arrest...
| Autores principales: | , , , , |
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| Formato: | Texto |
| Lenguaje: | English |
| Publicado: |
American Society for Biochemistry and Molecular Biology
2009
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| Materias: | |
| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2659243/ https://www.ncbi.nlm.nih.gov/pubmed/19171933 http://dx.doi.org/10.1074/jbc.M808463200 |