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Mice Deficient in Ribosomal Protein S6 Phosphorylation Suffer from Muscle Weakness that Reflects a Growth Defect and Energy Deficit
BACKGROUND: Mice, whose ribosomal protein S6 cannot be phosphorylated due to replacement of all five phosphorylatable serine residues by alanines (rpS6(P−/−)), are viable and fertile. However, phenotypic characterization of these mice and embryo fibroblasts derived from them, has established the rol...
| Autores principales: | , , , , , , , , , |
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| Formato: | Texto |
| Lenguaje: | English |
| Publicado: |
Public Library of Science
2009
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| Materias: | |
| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2682700/ https://www.ncbi.nlm.nih.gov/pubmed/19479038 http://dx.doi.org/10.1371/journal.pone.0005618 |