Vaccinia-induced epidermal growth factor receptor-MEK signalling and the anti-apoptotic protein F1L synergize to suppress cell death during infection

F1L is a functional Bcl-2 homologue that inhibits apoptosis at the mitochondria during vaccinia infection. However, the extent and timing of cell death during ΔF1L virus infection suggest that additional viral effectors cooperate with F1L to limit apoptosis. Here we report that vaccinia growth facto...

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Detalles Bibliográficos
Autores principales: Postigo, Antonio, Martin, Morag C, Dodding, Mark P, Way, Michael
Formato: Texto
Lenguaje:English
Publicado: Blackwell Publishing Ltd 2009
Materias:
Original Articles
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2730480/
https://www.ncbi.nlm.nih.gov/pubmed/19388902
http://dx.doi.org/10.1111/j.1462-5822.2009.01327.x

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2730480/
https://www.ncbi.nlm.nih.gov/pubmed/19388902
http://dx.doi.org/10.1111/j.1462-5822.2009.01327.x

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