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Systemic properties of metabolic networks lead to an epistasis-based model for heterosis
The genetic and molecular approaches to heterosis usually do not rely on any model of the genotype–phenotype relationship. From the generalization of Kacser and Burns’ biochemical model for dominance and epistasis to networks with several variable enzymes, we hypothesized that metabolic heterosis co...
Autores principales: | , , |
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Formato: | Texto |
Lenguaje: | English |
Publicado: |
Springer-Verlag
2009
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2793392/ https://www.ncbi.nlm.nih.gov/pubmed/19916003 http://dx.doi.org/10.1007/s00122-009-1203-2 |