Tumor necrosis factor-α and apoptosis signal-regulating kinase 1 control reactive oxygen species release, mitochondrial autophagy and c-Jun N-terminal kinase/p38 phosphorylation during necrotizing enterocolitis

BACKGROUND: Oxidative stress and inflammation may contribute to the disruption of the protective gut barrier through various mechanisms; mitochondrial dysfunction resulting from inflammatory and oxidative injury may potentially be a significant source of apoptosis during necrotizing enterocolitis (N...

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Detalles Bibliográficos
Autores principales: Baregamian, Naira, song, Jun, Bailey, C. Eric, Papaconstantinou, John, Evers, B. Mark, Chung, Dai H
Formato: Texto
Lenguaje:English
Publicado: Landes Bioscience 2009
Materias:
Research Paper
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2835918/
https://www.ncbi.nlm.nih.gov/pubmed/20716917

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2835918/
https://www.ncbi.nlm.nih.gov/pubmed/20716917

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