Interplay between REST and nucleolin transcription factors: a key mechanism in the overexpression of genes upon increased phosphorylation

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Non-malignant cells can be transformed via the activation of kinases that control degradation of neural-restrictive silencer factor (REST). Here, we identify a mechanism that contributes to the activation of genes, expression of which is controlled by responsive elements containing overlapping bindi...

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Detalles Bibliográficos
Autores principales: Tediose, Teeo, Kolev, Martin, Sivasankar, Baalasubramanian, Brennan, Paul, Morgan, B. Paul, Donev, Rossen
Formato: Texto
Lenguaje:English
Publicado: Oxford University Press 2010
Materias:
Gene Regulation, Chromatin and Epigenetics
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2875004/
https://www.ncbi.nlm.nih.gov/pubmed/20100803
http://dx.doi.org/10.1093/nar/gkq013

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2875004/
https://www.ncbi.nlm.nih.gov/pubmed/20100803
http://dx.doi.org/10.1093/nar/gkq013

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