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A novel CD93 polymorphism in non-obese diabetic (NOD) and NZB/W F1 mice is linked to a CD4+ iNKT cell deficient state

In the present study, we characterize a polymorphism in the CD93 molecule, originally identified as the receptor for the C1q complement component (i.e., C1qRp, or AA4.1) in non-obese diabetic (NOD) mice. This allele carries a coding polymorphism in the first epidermal growth factor-like domain of CD...

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Detalles Bibliográficos
Autores principales: Zekavat, Ghazal, Mozaffari, Raha, Arias, Vanessa J., Rostami, Susan Y., Badkerhanian, Armen, Tenner, Andrea J., Nichols, Kim E., Naji, Ali, Noorchashm, Hooman
Formato: Texto
Lenguaje:English
Publicado: Springer-Verlag 2010
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2875467/
https://www.ncbi.nlm.nih.gov/pubmed/20387063
http://dx.doi.org/10.1007/s00251-010-0442-3