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Overexpression of STARD3 in human monocyte/macrophages induces an anti-atherogenic lipid phenotype
Dysregulated macrophage cholesterol homoeostasis lies at the heart of early and developing atheroma, and removal of excess cholesterol from macrophage foam cells, by efficient transport mechanisms, is central to stabilization and regression of atherosclerotic lesions. The present study demonstrates...
Autores principales: | , , , |
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Formato: | Texto |
Lenguaje: | English |
Publicado: |
Portland Press Ltd.
2010
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2891001/ https://www.ncbi.nlm.nih.gov/pubmed/20491656 http://dx.doi.org/10.1042/CS20100266 |