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Prolonged Aβ treatment leads to impairment in the ability of primary cortical neurons to maintain K(+ )and Ca(2+ )homeostasis
BACKGROUND: Alzheimer's disease (AD) is a progressive neurodegenerative disease, characterised by the formation of insoluble amyloidogenic plaques and neurofibrillary tangles. Beta amyloid (Aβ) peptide is one of the main constituents in Aβ plaques, and is thought to be a primary causative agent...
| Autores principales: | , , , |
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| Formato: | Texto |
| Lenguaje: | English |
| Publicado: |
BioMed Central
2010
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| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2927593/ https://www.ncbi.nlm.nih.gov/pubmed/20704753 http://dx.doi.org/10.1186/1750-1326-5-30 |