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More severe cellular phenotype in human idiopathic dilated cardiomyopathy compared to ischemic heart disease
Activation of the β-adrenergic receptor (βAR) pathway is the main mechanism of the heart to increase cardiac output via protein kinase A (PKA)-mediated phosphorylation of cellular target proteins, and perturbations therein may contribute to cardiac dysfunction in heart failure. In the present study...
Autores principales: | , , , , , , , , , , , |
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Formato: | Texto |
Lenguaje: | English |
Publicado: |
Springer Netherlands
2010
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3005110/ https://www.ncbi.nlm.nih.gov/pubmed/21132354 http://dx.doi.org/10.1007/s10974-010-9231-8 |