Cargando…
Interleukin 1 receptor signaling regulates DUBA expression and facilitates Toll-like receptor 9–driven antiinflammatory cytokine production
The interleukin 1 receptor (IL-1R) and the Toll-like receptors (TLRs) are highly homologous innate immune receptors that provide the first line of defense against infection. We show that IL-1R type I (IL-1RI) is essential for TLR9-dependent activation of tumor necrosis factor receptor-associated fac...
Autores principales: | González-Navajas, Jose M., Law, Jason, Nguyen, Kim Phung, Bhargava, Meha, Corr, Mary Patricia, Varki, Nissi, Eckmann, Lars, Hoffman, Hal M., Lee, Jongdae, Raz, Eyal |
---|---|
Formato: | Texto |
Lenguaje: | English |
Publicado: |
The Rockefeller University Press
2010
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3005235/ https://www.ncbi.nlm.nih.gov/pubmed/21115691 http://dx.doi.org/10.1084/jem.20101326 |
Ejemplares similares
-
Interleukin 1 Receptor Dependence of Serum Transferred Arthritis Can be Circumvented by Toll-like Receptor 4 Signaling
por: Choe, Jung-Yoon, et al.
Publicado: (2003) -
Forward genetic analysis of Toll-like receptor responses in wild-derived mice reveals a novel antiinflammatory role for IRAK1BP1
por: Conner, James R., et al.
Publicado: (2008) -
ERK activation drives intestinal tumorigenesis in Apc(min/)(+) mice
por: Lee, Sung Hee, et al.
Publicado: (2010) -
Estrogen promotes cutaneous wound healing via estrogen receptor β independent of its antiinflammatory activities
por: Campbell, Laura, et al.
Publicado: (2010) -
Stimulation of Toll-like receptor 3 and 4 induces interleukin-1β maturation by caspase-8
por: Maelfait, Jonathan, et al.
Publicado: (2008)