Cargando…
Entrapment of Viral Capsids in Nuclear PML Cages Is an Intrinsic Antiviral Host Defense against Varicella-Zoster Virus
The herpesviruses, like most other DNA viruses, replicate in the host cell nucleus. Subnuclear domains known as promyelocytic leukemia protein nuclear bodies (PML-NBs), or ND10 bodies, have been implicated in restricting early herpesviral gene expression. These viruses have evolved countermeasures t...
Autores principales: | Reichelt, Mike, Wang, Li, Sommer, Marvin, Perrino, John, Nour, Adel M., Sen, Nandini, Baiker, Armin, Zerboni, Leigh, Arvin, Ann M. |
---|---|
Formato: | Texto |
Lenguaje: | English |
Publicado: |
Public Library of Science
2011
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3033373/ https://www.ncbi.nlm.nih.gov/pubmed/21304940 http://dx.doi.org/10.1371/journal.ppat.1001266 |
Ejemplares similares
-
Disruption of PML Nuclear Bodies Is Mediated by ORF61 SUMO-Interacting Motifs and Required for Varicella-Zoster Virus Pathogenesis in Skin
por: Wang, Li, et al.
Publicado: (2011) -
Neuronal Subtype and Satellite Cell Tropism Are Determinants of Varicella-Zoster Virus Virulence in Human Dorsal Root Ganglia Xenografts In Vivo
por: Zerboni, Leigh, et al.
Publicado: (2015) -
Current In Vivo Models of Varicella-Zoster Virus Neurotropism
por: Mahalingam, Ravi, et al.
Publicado: (2019) -
Varicella-Zoster Virus Transfer to Skin by T Cells and Modulation of Viral Replication by Epidermal Cell Interferon-α
por: Ku, Chia-Chi, et al.
Publicado: (2004) -
Varicella-Zoster Virus Infection Triggers Formation of an Interleukin-1β (IL-1β)-processing Inflammasome Complex
por: Nour, Adel M., et al.
Publicado: (2011)