AGEs Decrease Insulin Synthesis in Pancreatic β-Cell by Repressing Pdx-1 Protein Expression at the Post-Translational Level

Advanced glycation end products (AGEs) have been implicated in diverse pathological settings of many diabetic complications, and the possible mechanisms have been widely reported. However, the relationship between AGEs and pancreatic β-cell dysfunction is still poorly understood. Recent studies have...

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Detalles Bibliográficos
Autores principales: Shu, Tingting, Zhu, Yunxia, Wang, Hongdong, Lin, Yan, Ma, Zhuo, Han, Xiao
Formato: Texto
Lenguaje:English
Publicado: Public Library of Science 2011
Materias:
Research Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3078922/
https://www.ncbi.nlm.nih.gov/pubmed/21533167
http://dx.doi.org/10.1371/journal.pone.0018782

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3078922/
https://www.ncbi.nlm.nih.gov/pubmed/21533167
http://dx.doi.org/10.1371/journal.pone.0018782

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