Early detachment of neuromuscular junction proteins in ALS mice with SODG93A mutation

The transgenic animals with mutant copper/zinc superoxide dismutase (SOD1) DNA develop paralytic motor neuron disease resembling human amyotrophic lateral sclerosis (ALS) patients and are commonly used as models for ALS. In the transgenic (Tg) mice with the G93A mutation of the human SOD1 gene SOD1(...

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Detalles Bibliográficos
Autores principales: Narai, Hisashi, Manabe, Yasuhiro, Nagai, Makiko, Nagano, Isao, Ohta, Yasuyuki, Murakami, Tetsuro, Takehisa, Yasushi, Kamiya, Tatsushi, Abe, Koji
Formato: Texto
Lenguaje:English
Publicado: PAGEPress Publications 2009
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3093225/
https://www.ncbi.nlm.nih.gov/pubmed/21577353
http://dx.doi.org/10.4081/ni.2009.e16

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3093225/
https://www.ncbi.nlm.nih.gov/pubmed/21577353
http://dx.doi.org/10.4081/ni.2009.e16

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