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Btn3 is a negative regulator of Btn2-mediated endosomal protein trafficking and prion curing in yeast

Yeast Btn2 facilitates the retrieval of specific proteins from late endosomes (LEs) to the Golgi, a process that may be adversely affected in Batten disease patients. We isolated the putative yeast orthologue of a human complex I deficiency gene, designated here as BTN3, as encoding a Btn2-interacti...

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Detalles Bibliográficos
Autores principales: Kanneganti, Vydehi, Kama, Rachel, Gerst, Jeffrey E.
Formato: Texto
Lenguaje:English
Publicado: The American Society for Cell Biology 2011
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3093318/
https://www.ncbi.nlm.nih.gov/pubmed/21441304
http://dx.doi.org/10.1091/mbc.E10-11-0878