CCR9-CCL25 interactions promote cisplatin resistance in breast cancer cell through Akt activation in a PI3K-dependent and FAK-independent fashion

BACKGROUND: Chemotherapy heavily relies on apoptosis to kill breast cancer (BrCa) cells. Many breast tumors respond to chemotherapy, but cells that survive this initial response gain resistance to subsequent treatments. This leads to aggressive cell variants with an enhanced ability to migrate, inva...

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Detalles Bibliográficos
Autores principales: Johnson-Holiday, Crystal, Singh, Rajesh, Johnson, Erica L, Grizzle, William E, Lillard, James W, Singh, Shailesh
Formato: Online Artículo Texto
Lenguaje:English
Publicado: BioMed Central 2011
Materias:
Research
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3110128/
https://www.ncbi.nlm.nih.gov/pubmed/21539750
http://dx.doi.org/10.1186/1477-7819-9-46

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3110128/
https://www.ncbi.nlm.nih.gov/pubmed/21539750
http://dx.doi.org/10.1186/1477-7819-9-46

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