Cargando…
Endothelial and Macrophage-Specific Deficiency of P38α MAPK Does Not Affect the Pathogenesis of Atherosclerosis in ApoE(−/−) Mice
BACKGROUND: The p38α Mitogen-Activated Protein Kinase (MAPK) regulates stress- and inflammation-induced cellular responses. Factors implicated in the development of atherosclerosis including modified low-density lipoprotein (LDL), cytokines and even shear stress induce p38 activation in endothelial...
Autores principales: | Kardakaris, Rozina, Gareus, Ralph, Xanthoulea, Sofia, Pasparakis, Manolis |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Public Library of Science
2011
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3111465/ https://www.ncbi.nlm.nih.gov/pubmed/21695272 http://dx.doi.org/10.1371/journal.pone.0021055 |
Ejemplares similares
-
Smooth muscle cell specific NEMO deficiency inhibits atherosclerosis in ApoE(−/−) mice
por: Imai, Takashi, et al.
Publicado: (2022) -
Hematopoietic stem cell quiescence and function are controlled by the CYLD–TRAF2–p38MAPK pathway
por: Tesio, Melania, et al.
Publicado: (2015) -
TAP1-Deficiency Does Not Alter Atherosclerosis Development in Apoe
(−/−) Mice
por: Kolbus, Daniel, et al.
Publicado: (2012) -
Tumor Necrosis Factor (TNF) Receptor Shedding Controls Thresholds of Innate Immune Activation That Balance Opposing TNF Functions in Infectious and Inflammatory Diseases
por: Xanthoulea, Sofia, et al.
Publicado: (2004) -
Inhibitory effects of Mycoepoxydiene on macrophage foam cell formation and atherosclerosis in ApoE-deficient mice
por: Xia, Xiaochun, et al.
Publicado: (2015)