Cargando…
ER stress drives Lipocalin 2 upregulation in prostate cancer cells in an NF-κB-dependent manner
BACKGROUND: Tumor cells adapt to endoplasmic reticulum (ER) stress through a set of conserved intracellular pathways, as part of a process termed the unfolded protein response (UPR). The expression of UPR genes/proteins correlates with increasing progression and poor clinical outcome of several tumo...
Autores principales: | Mahadevan, Navin R, Rodvold, Jeffrey, Almanza, Gonzalo, Pérez, Antonio Fernández, Wheeler, Matthew C, Zanetti, Maurizio |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
BioMed Central
2011
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3146445/ https://www.ncbi.nlm.nih.gov/pubmed/21649922 http://dx.doi.org/10.1186/1471-2407-11-229 |
Ejemplares similares
-
Prostate cancer cells undergoing ER stress in vitro and in vivo activate transcription of pro-inflammatory cytokines
por: Mahadevan, Navin R, et al.
Publicado: (2010) -
Cell-Extrinsic Effects of Tumor ER Stress Imprint Myeloid Dendritic Cells and Impair CD8(+) T Cell Priming
por: Mahadevan, Navin R., et al.
Publicado: (2012) -
A Janus-faced role of the unfolded protein response in antitumor immunity
por: Mahadevan, Navin R., et al.
Publicado: (2013) -
Extracellular vesicles produced in B cells deliver tumor suppressor miR-335 to breast cancer cells disrupting oncogenic programming in vitro and in vivo
por: Almanza, Gonzalo, et al.
Publicado: (2018) -
A community affair in the tumor microenvironment
por: Rodvold, Jeffrey J., et al.
Publicado: (2017)