Cargando…
Accelerated Vascular Aging in CuZnSOD-Deficient Mice: Impact on EPC Function and Reparative Neovascularization
OBJECTIVE: Aging is associated with increased oxidative stress levels and impaired neovascularization following ischemia. CuZnSOD has an important role to limit oxidative stress in the vasculature. Here we investigated the role of CuZnSOD for the modulation of ischemia-induced neovascularisation dur...
Autores principales: | Groleau, Jessika, Dussault, Sylvie, Turgeon, Julie, Haddad, Paola, Rivard, Alain |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Public Library of Science
2011
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3155535/ https://www.ncbi.nlm.nih.gov/pubmed/21858065 http://dx.doi.org/10.1371/journal.pone.0023308 |
Ejemplares similares
-
Neuron specific reduction in CuZnSOD is not sufficient to initiate a full sarcopenia phenotype
por: Sataranatarajan, Kavithalakshmi, et al.
Publicado: (2015) -
Neuron‐specific deletion of CuZnSOD leads to an advanced sarcopenic phenotype in older mice
por: Bhaskaran, Shylesh, et al.
Publicado: (2020) -
Deletion of Neuronal CuZnSOD Accelerates Age-Associated Muscle Mitochondria and Calcium Handling Dysfunction That Is Independent of Denervation and Precedes Sarcopenia
por: Su, Yu, et al.
Publicado: (2021) -
The Lack of CuZnSOD Leads to Impaired Neurotransmitter Release, Neuromuscular Junction Destabilization and Reduced Muscle Strength in Mice
por: Shi, Yun, et al.
Publicado: (2014) -
Reduced expression of let‐7f activates TGF‐β/ALK5 pathway and leads to impaired ischaemia‐induced neovascularization after cigarette smoke exposure
por: Dhahri, Wahiba, et al.
Publicado: (2017)