Cargando…
miR-122 activates hepatitis C virus translation by a specialized mechanism requiring particular RNA components
In animals, microRNAs (miRNAs) generally repress gene expression by binding to sites in the 3′-untranslated region (UTR) of target mRNAs. miRNAs have also been reported to repress or activate gene expression by binding to 5′-UTR sites, but the extent of such regulation and the factors that govern th...
Autores principales: | Roberts, Ashley P. E., Lewis, Andrew P., Jopling, Catherine L. |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Oxford University Press
2011
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3177192/ https://www.ncbi.nlm.nih.gov/pubmed/21653556 http://dx.doi.org/10.1093/nar/gkr426 |
Ejemplares similares
-
The P body protein LSm1 contributes to stimulation of hepatitis C virus translation, but not replication, by microRNA-122
por: Roberts, Ashley P. E., et al.
Publicado: (2014) -
Eukaryotic translation initiation factor 4AII contributes to microRNA-122 regulation of hepatitis C virus replication
por: Ahmed, Choudhary Shoaib, et al.
Publicado: (2018) -
Liver-specific microRNA-122: Biogenesis and function
por: Jopling, Catherine
Publicado: (2012) -
Chemical structure requirements and cellular targeting of microRNA-122 by peptide nucleic acids anti-miRs
por: Torres, Adrian G., et al.
Publicado: (2012) -
Miravirsen (SPC3649) can inhibit the biogenesis of miR-122
por: Gebert, Luca F. R., et al.
Publicado: (2014)