Cargando…
Glutamate Release by Primary Brain Tumors Induces Epileptic Activity
Epileptic seizures are a common and poorly understood co-morbidity for individuals with primary brain tumors. To investigate peritumoral seizure etiology, we implanted patient-derived glioma cells into scid mice. Within 14–18 days, glioma-bearing animals developed spontaneous, recurring abnormal EEG...
Autores principales: | Buckingham, Susan C., Campbell, Susan L., Haas, Brian R., Montana, Vedrana, Robel, Stefanie, Ogunrinu, Toyin, Sontheimer, Harald |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
2011
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3192231/ https://www.ncbi.nlm.nih.gov/pubmed/21909104 http://dx.doi.org/10.1038/nm.2453 |
Ejemplares similares
-
Potassium and glutamate transport is impaired in scar-forming tumor-associated astrocytes
por: Campbell, Susan C., et al.
Publicado: (2019) -
Glutamate and tumor-associated epilepsy
por: Sontheimer, Harald
Publicado: (2011) -
Pathological Role for Exocytotic Glutamate Release from Astrocytes in Hepatic Encephalopathy
por: Montana, Vedrana, et al.
Publicado: (2014) -
Dysregulation of Ambient Glutamate and Glutamate Receptors in Epilepsy: An Astrocytic Perspective
por: Alcoreza, Oscar B., et al.
Publicado: (2021) -
Disruption of astrocyte-vascular coupling and the blood-brain barrier by invading glioma cells
por: Watkins, Stacey, et al.
Publicado: (2014)