Endothelial NO/cGMP/VASP Signaling Attenuates Kupffer Cell Activation and Hepatic Insulin Resistance Induced by High-Fat Feeding

OBJECTIVE: Proinflammatory activation of Kupffer cells is implicated in the effect of high-fat feeding to cause liver insulin resistance. We sought to determine whether reduced endothelial nitric oxide (NO) signaling contributes to the effect of high-fat feeding to increase hepatic inflammatory sign...

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Detalles Bibliográficos
Autores principales: Tateya, Sanshiro, Rizzo, Norma O., Handa, Priya, Cheng, Andrew M., Morgan-Stevenson, Vicki, Daum, Guenter, Clowes, Alexander W., Morton, Gregory J., Schwartz, Michael W., Kim, Francis
Formato: Online Artículo Texto
Lenguaje:English
Publicado: American Diabetes Association 2011
Materias:
Signal Transduction
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3198085/
https://www.ncbi.nlm.nih.gov/pubmed/21911751
http://dx.doi.org/10.2337/db11-0255

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3198085/
https://www.ncbi.nlm.nih.gov/pubmed/21911751
http://dx.doi.org/10.2337/db11-0255

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