Cargando…
Claudin-1 induced sealing of blood–brain barrier tight junctions ameliorates chronic experimental autoimmune encephalomyelitis
In experimental autoimmune encephalomyelitis (EAE), an animal model for multiple sclerosis (MS), loss of the blood–brain barrier (BBB) tight junction (TJ) protein claudin-3 correlates with immune cell infiltration into the CNS and BBB leakiness. Here we show that sealing BBB TJs by ectopic tetracycl...
Autores principales: | Pfeiffer, Friederike, Schäfer, Julia, Lyck, Ruth, Makrides, Victoria, Brunner, Sarah, Schaeren-Wiemers, Nicole, Deutsch, Urban, Engelhardt, Britta |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Springer-Verlag
2011
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3207130/ https://www.ncbi.nlm.nih.gov/pubmed/21983942 http://dx.doi.org/10.1007/s00401-011-0883-2 |
Ejemplares similares
-
Claudin-12 is not required for blood–brain barrier tight junction function
por: Castro Dias, Mariana, et al.
Publicado: (2019) -
DARC shuttles inflammatory chemokines across the blood–brain barrier during autoimmune central nervous system inflammation
por: Minten, Carsten, et al.
Publicado: (2014) -
Claudin interactions in and out of the tight junction
por: Van Itallie, Christina M, et al.
Publicado: (2013) -
Transcriptional Regulators of Claudins in Epithelial Tight Junctions
por: Khan, Niamat, et al.
Publicado: (2015) -
E7 oncoprotein from human papillomavirus 16 alters claudins expression and the sealing of epithelial tight junctions
por: Uc, Perla Yaceli, et al.
Publicado: (2020)